scubasean,
I don't have any research to back up those limits. I arrived at them because they seemed ulta conservative to me and I didn't like the idea of putting my son at risk. Those limits seemed, to me, to have a very low risk.
Neil,
You are in good company with your opinion on age 18, check out the article by Dr. Taylor.
I agree completely on SASY.
Genesis,
"I have been unable to find....
any solid science linking hyperbaric exposure to bone necrosis"
Where did you look?
Long-term Effects of Diving
Ernest S. Campbell, MD, FACS, Orange Beach, Ala.
[Medscape Orthopedics & Sports Medicine 2(5), 1998. © 1998 Medscape, Inc.]
"Dysbaric Osteonecrosis
Early in this century thousands of men were employed in the building of tunnels and bridges using compressed air to keep the workplace dry. It is from this population that the first reports of
disabling hip and shoulder conditions were verified radiographically as joint degeneration. The insidious nature of this condition can result in considerable bone damage prior to detection. In 1972, Edmonds and Thomas[5] estimated the incidence of dysbaric osteonecrosis was as high as 50% in divers. Ten case studies over the next ten years of divers who sought treatment for persistent joint pain were found to have osteonecrosis. The validity of both the Edmonds and Thomas results and some of the case studies are now being questioned because there was no established standard for radiologic diagnosis of dysbaric osteonecrosis.
Dysbaric osteonecrosis involves infarction of an area of bone due to the obstruction of terminal vessels of the bone's vascular supply, probably by gas emboli. The condition is thought to be a late manifestation of decompression sickness, frequent exposure to increased pressure, insufficient decompression on ascent, or inadequate treatment of decompression illness. Early diagnosis is by radiographic examination[6], scintography[7], MRI, and more recently ultrasonography.[8]
Osteonecrosis in divers presents in two basic forms: juxta-articular (subchondral), and shaft, which includes the neck and a portion of the long bone. The shaft lesions are predominately saponified fat, are usually asymptomatic, and are seldom of orthopedic significance. The juxta-articular lesions are of greater clinical significance, causing symptoms that are potentially disabling. These lesions show areas of dead bone surrounded by a layer of collagen which forms a fibrous band and new bone. Beyond is an area of creeping substitution and healing trabeculae (Fig. 4).
Frequently there is pain over the joint which may be aggravated by movement and radiate down the limb, and a slight restriction of movement is common. In the shoulder, the signs mimic rotator
cuff lesions, with pain from 60 to 180 degrees abduction and difficulty maintaining abduction against resistance. Following collapse of the cartilage, secondary degenerative arthritis develops with further reduction in joint motion. The femur is affected two to three times more often than the humerus.
Imaging
The radiograph is the gold standard for diagnosing dysbaric osteonecrosis but it depends on the quality of the radiograph and the radiologist's experience. Although only the shoulders and the hips are affected, extensive views of the lower femur and upper tibia are included to identify as many shaft lesions as possible.
The incidence of avascular necrosis in the general population is unknown, so the alternative causes of bone necrosis should be excluded when the condition is found in divers. They include
hyperlipidemia, diabetes mellitus, pancreatitis, cirrhosis with chronic alcoholism, long-term steroid therapy, Gaucher's Disease, and other conditions that may be incompatible with fitness for diving.
Although the diagnostic standard, radiography is not a good tool to demonstrate changes over time. Other techniques are available and have value in screening for the disease. MDP
(99mTechnetium Methyl-dipolyphosphate) scans are very sensitive to local bone pathology. A "hot spot" indicates increased perfusion and metabolism and changes are recognized only hours after a dive. A positive scan indicates a need for radiological follow-up and is not diagnostic.
Magnetic resonance imaging (MRI) (Fig. 4) has a remarkable power to detect early lesions but because of expense it is not generally available for routine screening of large populations. It was used in 1981 by the Decompression Sickness Registry, who found that the percentage of bone necrosis, both shaft and juxta-articular, increases in a sample of divers with age and experience. At least one definite lesion was found in 4.2% of a population of 4980 divers. Necrosis was not found in those who had never dived deeper than 30 meters, but was detected in 30 out of 190 men (15.8%) who had dived deeper than 200 meters. It can be argued that screening of deep divers with MRI can detect juxta-articular lesions and prevent joint collapse.[9]
Early recognition is imperative, and can be accomplished by annual long bone radiographic examinations, radiographic investigation of any minor arthralgia or bursitis, and follow-up
radiographs two months after a decompression episode. Asymptomatic lesions should restrict diving to shallow depths with proscription of decompression, experimental, and commercial diving. Obviously, juxta-articular lesions preclude any diving whatsoever. Early surgical treatment by decortication of the involved area with prosthesis is recommended."
The
NOAA Diving Manual also discusses the relationship on page 3-29.
The following is not something I would expect you to find. I saved it at the time. It is a personal account of Patti O'B. and was posted on the Rodale's forum in 1997. It is NOT solid scientific evidence, but I would need solid scientific evidence diving does not cause problems in a child's development before I'd change my position - children are too precious to risk.
"Posted by Patti O'B on November 16, 1997 at 11:40:05:
In Reply to: No deeper than 20m posted by CWS on November 14, 1997 at 14:36:35:
Age is not a good measure of maturity or physical ability, but there are many physiological reasons why you should read the medical literature and assess the risks. There are unique physical differences between growing bodies and adults. We still don't know so much about DCS. Read the literature on how the tables were created. Then take a look at the questions surrounding the effects of hormone levels, fat/muscle ratios, and age on gas exchange in the blood. Read up on the patent foramen debate. And read about the effects of pressure on bones.
And to reinforce this advice, here's a story ...
I started diving at age 11. There wasn't a JR card then, so I finished the advanced diver training which had no age restrictions and started diving fast and furiously. I dove with a terrific dive group, but I was the only one my age. I dove the group profiles of up to 150' feet. Within the first year I logged over 200 dives, then started assisting classes, taking more courses, and doing lots and lots of diving. It was wonderful! I loved every minute of it.
Then at 18, I discovered a medical problem- my bones and cartilege, primarily in my spine- were degenerating rapidly. No family history of this problem. So a surgery and some PT later, I was back in the swing of things, working now as an AI and studying for a degree in underwater archaelogy, working as a dig safety officer and generally in the water 5-6 days a week minimum 4 hours at a time. Whoops- more back problems (surprise, surprise) Final diagnosis was repeated stress injuries from heavy involvement in "high impact sports" such as gymnastics and yes, diving. Five years, three surgeries and countless hours of PT later, I regained most of my mobility. But- it meant finishing my degree from the hospital (not in archaelogy because it was fieldwork that appealed to me), missing out on most of my last two years of college, spending the four months after my wedding in yet another hospital, and now figuring out how to do everything with only partial use of one leg and a lifting restriction of 10-15 lbs!
I'm not saying the diving by itself caused this. But it is very likely that diving influenced the development of the bones and encouraged the degeneration (not just my opinion by also my diving orthopedic surgeon's and a host of physical therapists, doctors, nurses, techs ). Add to this the stress of lifting tanks, gear, etc which compounded the problems.
Take the time to read the literature (not just about the effects on bone but also about your heart), talk to a diving MD and then assess the risks. It isn't just about reaching your full height. You can be 6'3" and not have finished bone development. After knowing the potential risks, then you can make your own decision about profiles, frequency, etc. The literature is by no means cut and dried. But there is enough there (IMHO) to justify being cautious. You've got your whole life to dive, and though I can still dive- it certainly isn't as much fun as it used to be!
Will I let my kids dive? Unless the literature changes in the next ten years...Absolutely! Will I restrict that diving- you better believe it!!
Take care all- and dive safely!!
Patti O'B"
For a long discussion on a host of issues related to children and diving go to Larry "Harris" Taylor's discussion on
Why I do NOT Train Kids from Our World Underwater, April 28, 2001 in Chicago.
The bottom line is why take chances with the most precious things on this planet - our children.