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There are numerous reports of chronic lung damage and eye problems (retrolental fibroplasia) in laboratory animals and premature infants exposed to high levels of oxygen.
This does not seem to translate to the adult who is subjected to periodic hyperbaric oxygenation. However, Suzuki has reported in the Undersea Medical Journal in 1994 that in saturation divers HBO caused a decrease in diffusing capacity in the lungs and an increase in ethane production. These observations indicated that the decrease of DLCO/VA was assumed to be caused by oxygen-derived free radicals.
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