^ that's not true. You do not form clots in your extremities when you have normal vasoconstriction. That would be part of frostbite and reperfusion syndrome. Normal dives or being out in the cold normally does not cause a bunch of platelets to clot in your fingers/toes.
I will not comment on if there is a true benefit to using ASA in DCI as I'm not a dive physician but it seems like a quick lit search reveals it doesn't make a difference to preemptively take it.
Also ASA's mechanism of action is inhibition of platelet aggregation. It does not break up clots that may already exist in certain disease states. It does not "thin" the blood. It does not increase perfusion. It prevents new clots from forming via platelet aggregation (there are still other ways to form clots though). The theoretical benefit to giving ASA in suspected DCI is to inhibit clot formation secondary to platelet aggregation during an immune mediated response to bubbles, if you are a believer in that mechanism of DCI.
I will not comment on if there is a true benefit to using ASA in DCI as I'm not a dive physician but it seems like a quick lit search reveals it doesn't make a difference to preemptively take it.
Also ASA's mechanism of action is inhibition of platelet aggregation. It does not break up clots that may already exist in certain disease states. It does not "thin" the blood. It does not increase perfusion. It prevents new clots from forming via platelet aggregation (there are still other ways to form clots though). The theoretical benefit to giving ASA in suspected DCI is to inhibit clot formation secondary to platelet aggregation during an immune mediated response to bubbles, if you are a believer in that mechanism of DCI.