The Iceni
Medical Moderator
Originally posted by zelevin
So - let me see - is sneezing when getting out of water potentially dangerous as well for people with PFO? Ouch.
- Vladimir
Dr Vikingo is quite right to add the risks of a joint DCI to this discussion and it was remiss of me to leave it out.
Vladimir, You described a type II DCI, where you had quite alarming symtoms related to your central nervous system and to my mind this can only, commonly, occur in two situations.
1) A rapid ascent ( or an ascent with missed stops) which leads to the formation of bubble seeds in soft tissues themselves, or
2) Bubble nuclei from the venous circulation finding their way into the arterial circulation embolising to vital organs in which they lodge, then growing as they take on more gas at the surface. This can only occur when a right to left shunt exists .
Variations on the Valsalva manouevre performed by divers include forcefully clearing your ears, straining to carry heavy equipment ("grunting"), coughing and yes sneezing. I cannot imagine anything a human being could do that could cause a more explosive change to the intrathoracic pressure than to sneeze! A sneeze is produced by a rapid reflex contraction of the diaphragm and the muscles of the chest wall in order to to expel the contents of the nasal airways, followed by an immediate return to normal. So if anything could cause a bolus of blood to pass through any shunt a sneeze is first on my list.
By the way, A PFO is certainly not the only cause of type II DCI. I was recently contacted by a very experienced diver who had suffered a neurological DCI following a very leasurely dive to 20 metres (66 feet) for 18 minutes with a five miute safety stop at 6 metres. Personally I doubt this DCI was due to a shunt because, if he had a significant PFO I would have expected him to have suffered a DCI much earlier in his 16 year diving career and he had done nothing to precipitate such a DCI.
It transpired that he had suffered a prolapsed intervertebral disc some twenty years ago, which had caused spinal nerve compression at the time. He had to stay off work until he was cured by means of a spinal operation, a laminectomy.
I feel this may have been the culprit in his case, particularly as the distribution of the neurological deficite from the DCI mirrored exactly the side and the level of the damage from his original PID.
All operations produce potential spaces and the blood supply to scar tissue is very poor. We may never know the real culprit but if he is wise, I suspect this chap's diving career is over.
I wonder what Dr Vikingo or the other regulators make of this incident.